This patient support community is for discussions relating to angina, angioplasty, arrhythmia, bypass surgery, cardiomyopathy, coronary artery disease, defibrillator, heart attack, heart disease, high blood pressure, mitral valve, pacemaker, PAD, stenosis, and stress tests.
I have never smoked, never used alcohol, never used any drugs.
"The echo indicated mild asymmetric left ventricular hypertrophy, a mildly dilated right ventricle, an EF 55-60%, mild aortic regurgitation, mild mitral regurgitation and trace tricuspid regurgitation. There was no wall motion abnormalities noted. The aortic root diameter is 3.3 CM (down from 3.9.. which doesn't make sense)"
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The aortic root diameter is an estimate and usually the area closest to the valve is narrower. So the variation may be due to exactly the location measured, regardless it doesn't appear to be an issue.
A 64 slice CT angiogram takes images of the anatomy of the vessels, and for someone who has a family history, it seems that would be an appropriate test. A cath angiogram just views lumen and should see any large blockage.
Are you taking medication such as a beta blocker. That can cause some fatigue.
A faster beating heart may have induced ischemia and that can be an explanation regarding high sinus rhythm and slightly depressed ST on an EKG?!
I did do a Coronary CTA in May. I was trying to avoid a catherization to be honest!
The results showed a mild plaque burden of 50.
It stated that there was a small circumflex artery that very quickly leaves the AV groove and becomes a marginal branch to the left ventricle.
There were 2 very small peripheral calcified plaques in the proximal RCA.
It indicated a 7MM long, densely calcified plaque in the proximal LAD lumen, distal to the 1st diagonal branch, which obscured the lumen in the images.
That was disconcerting!
When the cath was done, the cardiologist said that the calcified plaque must be lying on the outside of the LAD arterial wall and that there were no blockages.
He also measured the pulmonary artery for hypertension and told me that I do not have pulmonary hypertension, which is great!
Initially, the cardiologist thought that I would be stented but once the procedure was underway, he soon assessed that it wasn't necessary.
My family was elated!
The cardiologist and his research assistant were very complementary of my proactive care which has included Lipitor for 10 years, zetia, flaxseed oil and a few other vitamins. My cholesterol levels are excellent. They felt that this has helped me despite my family history.
I am not on a beta blocker because I take allergy injections and beta blockers can not be used with them.
I am very concerned that I am unable to stand for more than a moment or two!
I do remember my mother having the red face with minimal exertion, the sweating, fast heartbeat and trembling.
I do know that I am fighting genetics